On 2016 Dec 12, David Mage commented:

Carlin and Moon have provided a very thorough review of risk factors for SIDS with one major flaw – a complete lack of recognition that SIDS have a male excess of 50% corresponding to a male fraction of 0.60, or 3 males for each 2 females. CDC (http://wonder.cdc.gov) reports for years 1968-2014 that for causes of death by SIDS, Unknown (UNK) and Accidental Suffocation and Strangulation in Bed (ASSB) there were 119,201 male and 79,629 female post-neonatal cases (28-364 days) for a male fraction of 0.600. Naeye et al. (PMID 5129451) were, to our knowledge, the first to claim that this 50% male excess in infant mortality must be X-linked. We agreed, and have proposed that an X-linked recessive allele with frequency q = 2/3 that is not protective against acute anoxic encephalopathy would place XY males at risk with frequency q = 2/3 and XX females at risk with frequency q*q = 4/9 (PMID 9076695, 15384886).

On 2016 Dec 12, David Mage commented:

Carlin and Moon have provided a very thorough review of risk factors for SIDS with one major flaw – a complete lack of recognition that SIDS have a male excess of 50% corresponding to a male fraction of 0.60, or 3 males for each 2 females. CDC (http://wonder.cdc.gov) reports for years 1968-2014 that for causes of death by SIDS, Unknown (UNK) and Accidental Suffocation and Strangulation in Bed (ASSB) there were 119,201 male and 79,629 female post-neonatal cases (28-364 days) for a male fraction of 0.600. Naeye et al. (PMID 5129451) were, to our knowledge, the first to claim that this 50% male excess in infant mortality must be X-linked. We agreed, and have proposed that an X-linked recessive allele with frequency q = 2/3 that is not protective against acute anoxic encephalopathy would place XY males at risk with frequency q = 2/3 and XX females at risk with frequency q*q = 4/9 (PMID 9076695, 15384886).