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  1. Last 7 days
  2. 54.241.53.176 54.241.53.176
    PMID_40950137-html.html
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    54.241.53.176/c1b38c9eb32a1a6793c175a37eaf05785834526a/PMID_40950137-html.html
  3. Sep 2025
  4. 54.241.53.176 54.241.53.176
    A double-assurance mechanism restrains generation of high potential transit-amplifying progenitors during neurogenesis
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    54.241.53.176/ff316ceeefba14c54f66a5fbcfae2e6952c4b24a/2025.09.16.676671v3.full-html.html
  5. Apr 2025
  6. pmc.ncbi.nlm.nih.gov pmc.ncbi.nlm.nih.gov
    A newly discovered neural stem cell population is generated by the optic lobe neuroepithelium during embryogenesis in Drosophila melanogaster
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    pmc.ncbi.nlm.nih.gov/articles/PMC6176933/
  7. Jul 2024
  8. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    The ETS-transcription factor Pointed is sufficient to regulate the posterior fate of the follicular epithelium
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    ncbi.nlm.nih.gov/pmc/articles/PMC7687869/
  9. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    Ratiometric sensing of Pnt and Yan transcription factor levels confers ultrasensitivity to photoreceptor fate transitions in Drosophila
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    ncbi.nlm.nih.gov/pmc/articles/PMC10163347/
  10. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    A single-cell atlas and lineage analysis of the adult Drosophila ovary
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    ncbi.nlm.nih.gov/pmc/articles/PMC7648648/
  11. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    A multi-gene knockdown approach reveals a new role for Pax6 in controlling organ number in Drosophila
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    ncbi.nlm.nih.gov/pmc/articles/PMC8172120/
  12. May 2024
  13. www.ncbi.nlm.nih.gov www.ncbi.nlm.nih.gov
    EGFR signaling controls directionality of epithelial multilayer formation upon loss of cell polarity
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    ncbi.nlm.nih.gov/pmc/articles/PMC10711663/
  14. Feb 2024
  15. elifesciences.org elifesciences.org
    Diversification of heart progenitor cells by EGF signaling and differential modulation of ETS protein activity
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    elifesciences.org/articles/32847
  16. Sep 2020
  17. elifesciences.org elifesciences.org
    Tailless/TLX reverts intermediate neural progenitors to stem cells driving tumourigenesis via repression of asense/ASCL1
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    elifesciences.org/articles/53377